Feno F

Mechanism of action of Rosuvastatin is that it inhibits __________enzyme and thereby inhibits cholesterol synthesis.

• HMGCoA Synthase

• HMGCoA Reductase

• ACL

• None of these

Fenofibrate is a ________agonist.

• PPAR Beta

• PPAR Gama

• PPAR Alpha

• None of these

Fenofibrate is instrumental in increasing ________enzyme which converts triglycerides to Free fatty acids and oxidation of free fatty acid

• Hepatic Lipase

• LpLipase

• Amylase

• Chymase

As per communication in VAF, rosuvastatin in Fortius reduces atherogenic index by _____%

• 10

• 9

• 7.11

• 9.11

As per current VAF, Fortius F is being promoted in _____________

• Hypercholesterolemia

• Hypertriglyceridemia

• Combined hyperlipidemia

• None of these

State True or False: Rosuvastatin and Fenofibrate combination

curtails oxidative stress markers

• True

• False

As per communication in VAF, Combined hyperlipidemia poses a ____fold higher CV risk 1 due to elevated LDL C, Triglycerides , and low HDL C levels .

• 5

• 7

• 9

• 10

Ideal level of triglycerides in human body should be _____mg/dl

• <50

• <90

• <150

• <200

Very high level of Triglycerides are defined as

• 200 mg/dl

• 200 to 499 mg/dl

• >500 mg/dl

• 150 to 300 mg/dl

Borderline level of Triglycerides are defined as

• <150 mg/dl

• 150 to 199 mg/dl

• 200 to 499 mg/dl

• >500 mg/dl